Sestrin2 Overexpression Inhibits Proliferation and Epithelial–Mesenchymal Transition and Induces Autophagy Through the AMPK/mTOR Signaling Pathway in Human Prostate Cancer Cells
Conclusion: Our study suggests that SESN2 inhibits prostate cancer cells by inducing autophagy through the AMPK/mTOR signaling pathway. These results indicate that SESN2 might be a novel target for prostate cancer.
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Main Authors: | , , , , |
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Format: | Article |
Language: | English |
Published: |
Wiley
2025-01-01
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Series: | Prostate Cancer |
Online Access: | http://dx.doi.org/10.1155/proc/8842203 |
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