Acute adrenaline myocardial necrosis prevention by artificial lung ventilation in experimental settings

Acute adrenaline myocardial necrosis prevention by artificial lung ventilation in experimental settings

In experimental settings, hystotoxic catecholamine doses depress cardiomyocyte contractility, induce arrhythmia, and heart arrest. Edema and hemorrhages in lung tissue and myocardium, myocardial hemomicrocirculation disturbances, significantly disturbed CO2, O2 partial pressure, and blood pH, plus i...

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Bibliographic Details
Main Authors: S. A. Sisakyan, N. F. Krasnikov, A. S. Sisakyan, A. B. Semerdzhyan, V. A. Oganyan
Format: Article
Language:Russian
Published: «FIRMA «SILICEA» LLC 2007-02-01
Series:Российский кардиологический журнал
Subjects:
catecholamines
toxic doses
negative effect
heart function
artificial lung ventilation
positive role
Online Access:https://russjcardiol.elpub.ru/jour/article/view/2048
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Summary:In experimental settings, hystotoxic catecholamine doses depress cardiomyocyte contractility, induce arrhythmia, and heart arrest. Edema and hemorrhages in lung tissue and myocardium, myocardial hemomicrocirculation disturbances, significantly disturbed CO2, O2 partial pressure, and blood pH, plus increased acidosis are observed. Artificial lung ventilation (ALV) prevents fibrillation and heart arrest, edema and generalized lung hemorrhages after lethal catecholamine dose infusion. In addition, ALV prevents hypercapnia, acidosis, and increases blood oxygenation.
ISSN:1560-4071
2618-7620

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