Αποτελέσματα αναζήτησης - Liyuan Wang

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  1. 1

    Mainlobe Jamming Suppression via Joint Polarization-Range-Doppler Processing από Liyuan Wang, Huafeng He, Zhen Li, Buma Xiao, Tao Zhou

    Έκδοση 2025-06-01

    In the field of electromagnetic countermeasures, suppressing mainlobe jamming represents a critical challenge requiring urgent resolution. Conventional polarization-based anti-jamming techniques, which fundamentally rely on obtaining pure jamming signals for prior parameter estimation, demonstrate l...

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  2. 2

    Single-Cell Atlas of Spleen Remodeling Reveals Macrophage Subset-Driven ASFV Pathogenesis από Liyuan Wang, Shouzhang Sun, Lei Liu, Yun Chen, Haixue Zheng, Zhonglin Tang

    Έκδοση 2025-07-01

    African swine fever virus (ASFV) causes global swine outbreaks, but its cellular pathogenesis is poorly understood. Using single-cell RNA data from ASFV-infected pig spleens across four timepoints, we identified macrophages as the primary viral reservoir, with infection driving lymphoid depletion an...

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  3. 3

    Systemic inflammatory and oxidative stress biomarkers as predictors of pain in sequential bilateral phacoemulsification for age-related cataract από Peng Yin, Peng Yin, Liyuan Wang, Yongsheng Hou, Yang Huang, Dawei Sun, Dewang Shao

    Έκδοση 2025-07-01

    BackgroundSecond-eye cataract surgery is frequently associated with enhanced pain perception compared to first-eye procedures, yet the underlying pathophysiological mechanisms remain incompletely understood. This study investigated whether systemic inflammatory biomarkers and oxidative stress indica...

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  4. 4

    Case Report: Successful treatment of spondyloenchondrodysplasia with immune dysregulation using tofacitinib and ruxolitinib: a report of two pediatric cases από Chengzhu Liu, Zhiwei Xie, Min Wang, Jinhua Chu, Linhai Yang, Kunlong Zhang, Lingling Huang, Songji Tu, Huaju Cai, Zhengyu Wu, Liyuan Wang, Ningling Wang

    Έκδοση 2025-07-01

    Spondyloenchondrodysplasia with immune dysregulation (SPENCDI) is autosomal recessive hereditary disease caused by tartrate resistant acid phosphatase 5 (ACP5) mutations. The symptoms mainly involve the bone, immune system and nervous system, and the typical manifestations are short stature, abnorma...

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