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    NDP52 deficiency accelerates chondrocyte degeneration through promoting pathogenic mitochondrial ROS via reverse electron transport

    NDP52 deficiency accelerates chondrocyte degeneration through promoting pathogenic mitochondrial ROS via reverse electron transport von Yutao Zhu, Yaohan Xu, Dinqi Xie, Nengfeng Yu, Jiaxin Chen, Jiechao Xia, Zixuan Mei, Yang Jin, Chuan Hu, Pan Tang, Sicheng Jiang, Chao Jiang, Honghai Song, Zhijun Hu

    Veröffentlicht 2025-09-01

    NDP52, a constituent of the selective autophagy receptors (SARs), was recognized for its involvement in facilitating substrate degradation via autophagic bridging. However, its autonomous function apart from autophagy remained largely unexplored. Here, we reported that NDP52 was down-regulated in de...

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